What is the science on "Digestibility Correct" foods?
https://wiodiet.com/ foods have 2 separate nutritional labels: * Regular Nutritional Facts - E.g. 500 Cal for a Croissant * Digestibility Corrected Nutritional Facts - 50 Cal for the same Croissant There alleged way this works is by having some enzyme inhibitors so your body wouldn't digest most of the food. I can see a lot of articles on these inhibitors like Amylase inhibitors, but I can't infer the safety or efficacy of these foods like that. I have tried their croissant myself and it is really good, but it does sound too good to be true. Looking up online I can find opinions of Ketonistas that had success and failure with these foods.
Hypertension
Hi Peter and team, Could you please consider some further deep dives on hypertension that launch from last week’s episode w Ethan Weiss. It would be great if you could give Blood Pressure your in depth treatment, much like lipids and insulin resistance. Thank you for all you do!
Is there a general consensus with how ApoB results are interpreted?
So I've been following your podcast for a while, and I couldn't agree more with importance of ApoB as a biomarker to diagnose CVD risk. While ApoB is relatively new to many HCPs and potentially more costly than the standard lipid profile, are there any other reasons as to why its not commonly used? For example, is there a lack of a standardised measuring method which complicates clinical interpretation? (ChatGPT came up with this potential reason, but I can't seem to find any mention of this in the literature).
Problematic high LDL
Indeed these are three related but separate questions related to LDL. So, Peter has been very clear that what he sees from all the scientific evidence related to high LDL is that high LDL is a necessary factor for CVD. So a life with low LDL is better to promote health. So, my questions are: - How to reduce LDL only with diet and exercise? Or saying it in other words, How is LDL coming up in and individual with metabolic syndrome? Is LDL totally uncoupled to metabolic syndrome? Basically, what I'm trying to query here is, how a person diet and lifestyle induce high LDL? - Now, in a "healthy" person where all biomarkers are in the right place except LDL (like a lean mass hyper responder), what is creating high LDL? How to reduce LDL without medicine but with exercise + a diet (not necessary keto) that keeps the rest of the biomarkers in check? - Can you provide the references to the scientific articles that show that hyper-responders on a keto diet + exercise with all biomarkers in check except LDL are in risk of CVD? I mean papers responding to this exact group of people (not other pieces of the puzzle to infer the risk, but clear specific scientific evidence of hyper responders under keto + exercise. Thanks